Reactome: A Curated Pathway Database

Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

If you have an ORCID ID that is not listed on this page, please forward this information to us and we will update your Reactome pathway records.

Name Email address

Pathways reviewed by Lindahl, Tomas (111150)

DB_ID Name
73894 DNA Repair

Details on Person Lindahl, Tomas

Class:IdPerson:111150
_displayNameLindahl, Tomas
_timestamp2017-08-22 20:37:27
affiliation[Affiliation:1243088] Cancer Research UK, London Research Institute
firstnameTomas
initialT
modified[InstanceEdit:418618] D'Eustachio, P, 2009-04-24 15:53:07
[InstanceEdit:1243087] D'Eustachio, P, 2011-03-31
[InstanceEdit:8932050] D'Eustachio, Peter, 2016-07-20
surnameLindahl
(author)[LiteratureReference:110557] Quality control by DNA repair
[LiteratureReference:111248] Second pathway for completion of human DNA base excision-repair: reconstitution with purified proteins and requirement for DNase IV (FEN1).
[LiteratureReference:111257] Reconstitution of DNA base excision-repair with purified human proteins: interaction between DNA polymerase beta and the XRCC1 protein.
[LiteratureReference:113684] Reversal of DNA alkylation damage by two human dioxygenases.
[LiteratureReference:113692] Oxidative demethylation by Escherichia coli AlkB directly reverts DNA base damage.
[LiteratureReference:3247931] Mutations in the gene encoding the 3'-5' DNA exonuclease TREX1 cause Aicardi-Goutières syndrome at the AGS1 locus
[LiteratureReference:3248000] Heterozygous mutations in TREX1 cause familial chilblain lupus and dominant Aicardi-Goutieres syndrome
[LiteratureReference:8857712] N6-methyladenosine in nuclear RNA is a major substrate of the obesity-associated FTO
[InstanceEdit:165097] Khanna, KK, Lindahl, T, West, SC, Wood, Richard D, 2004-06-01
[Change default viewing format]