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Pathways reviewed by Lindahl, Tomas (111150)
Details on Person Lindahl, Tomas
|affiliation||[Affiliation:1243088] Cancer Research UK, London Research Institute|
|modified||[InstanceEdit:418618] D'Eustachio, P, 2009-04-24 15:53:07|
[InstanceEdit:1243087] D'Eustachio, P, 2011-03-31
[InstanceEdit:8932050] D'Eustachio, Peter, 2016-07-20
|(author)||[LiteratureReference:110557] Quality control by DNA repair|
[LiteratureReference:111248] Second pathway for completion of human DNA base excision-repair: reconstitution with purified proteins and requirement for DNase IV (FEN1).
[LiteratureReference:111257] Reconstitution of DNA base excision-repair with purified human proteins: interaction between DNA polymerase beta and the XRCC1 protein.
[LiteratureReference:113684] Reversal of DNA alkylation damage by two human dioxygenases.
[LiteratureReference:113692] Oxidative demethylation by Escherichia coli AlkB directly reverts DNA base damage.
[LiteratureReference:3247931] Mutations in the gene encoding the 3'-5' DNA exonuclease TREX1 cause Aicardi-Goutières syndrome at the AGS1 locus
[LiteratureReference:3248000] Heterozygous mutations in TREX1 cause familial chilblain lupus and dominant Aicardi-Goutieres syndrome
[LiteratureReference:8857712] N6-methyladenosine in nuclear RNA is a major substrate of the obesity-associated FTO
[InstanceEdit:165097] Khanna, KK, Lindahl, T, West, SC, Wood, Richard D, 2004-06-01
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