Reactome: A Curated Pathway Database

Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

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Pathways authored by Shah, Kavita (3301955)

DB_ID Name
8862803 Deregulated CDK5 triggers multiple neurodegenerative pathways in Alzheimer's disease models

Pathways reviewed by Shah, Kavita (3301955)

DB_ID Name
8854521 Interaction between PHLDA1 and AURKA

Details on Person Shah, Kavita

Class:IdPerson:3301955
_displayNameShah, Kavita
_timestamp2017-08-22 20:37:50
affiliation[Affiliation:8855529] Department of Chemistry, Purdue University
created[InstanceEdit:3302058] Jassal, B, 2013-04-23
firstnameKavita
initialK
modified[InstanceEdit:8855531] Orlic-Milacic, Marija, 2016-02-04
[InstanceEdit:8932050] D'Eustachio, Peter, 2016-07-20
surnameShah
(author)[InstanceEdit:8855530] Shah, Kavita, 2016-02-04
[InstanceEdit:8862798] Shah, Kavita, 2016-02-23
[LiteratureReference:3301965] Glutathione-S-transferase P1 is a critical regulator of Cdk5 kinase activity
[LiteratureReference:8853426] PHLDA1 is a crucial negative regulator and effector of Aurora A kinase in breast cancer
[LiteratureReference:8862800] Deregulated Cdk5 triggers aberrant activation of cell cycle kinases and phosphatases inducing neuronal death
[LiteratureReference:8868146] Nuclear envelope dispersion triggered by deregulated Cdk5 precedes neuronal death
[LiteratureReference:8868261] Novel genetic tools reveal Cdk5's major role in Golgi fragmentation in Alzheimer's disease
[LiteratureReference:8868566] Deregulated Cdk5 promotes oxidative stress and mitochondrial dysfunction
[LiteratureReference:8868669] Cdk5 is a major regulator of p38 cascade: relevance to neurotoxicity in Alzheimer's disease
[LiteratureReference:8868671] Direct and indirect roles of cyclin-dependent kinase 5 as an upstream regulator in the c-Jun NH2-terminal kinase cascade: relevance to neurotoxic insults in Alzheimer's disease
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