Reactome: A Curated Pathway Database

Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

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Pathways reviewed by Wajant, Harald (3465486)

DB_ID Name
75893 TNF signaling
5357786 TNFR1-induced proapoptotic signaling
5357905 Regulation of TNFR1 signaling
5357956 TNFR1-induced NFkappaB signaling pathway
5626978 TNFR1-mediated ceramide production

Details on Person Wajant, Harald

Class:IdPerson:3465486
_displayNameWajant, Harald
_timestamp2017-08-22 20:39:48
created[InstanceEdit:3465480] Shamovsky, V, 2013-05-17
firstnameHarald
initialH
surnameWajant
(author)[LiteratureReference:3465396] Selective inhibition of FLICE-like inhibitory protein expression with small interfering RNA oligonucleotides is sufficient to sensitize tumor cells for TRAIL-induced apoptosis
[LiteratureReference:3465476] NFkappaB activation by Fas is mediated through FADD, caspase-8, and RIP and is inhibited by FLIP
[LiteratureReference:5634170] Caspase-mediated cleavage converts the tumor necrosis factor (TNF) receptor-associated factor (TRAF)-1 from a selective modulator of TNF receptor signaling to a general inhibitor of NF-kappaB activation
[LiteratureReference:5657935] Tumor necrosis factor receptor-associated factor (TRAF) 1 regulates CD40-induced TRAF2-mediated NF-kappaB activation
[LiteratureReference:5668474] Membrane tumor necrosis factor (TNF) induces p100 processing via TNF receptor-2 (TNFR2)
[LiteratureReference:5692375] Sustained JNK activation in response to tumor necrosis factor is mediated by caspases in a cell type-specific manner
[InstanceEdit:6792551] Wajant, Harald, 2015-08-25
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