Reactome: A Curated Pathway Database

Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

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Name Email address

Pathways reviewed by Tolan, Dean R (352934)

DB_ID Name
5657562 Essential fructosuria
5657560 Hereditary fructose intolerance

Details on Person Tolan, Dean R

Class:IdPerson:352934
_displayNameTolan, Dean R
_timestamp2017-08-22 20:39:57
affiliation[Affiliation:5675802] Biology Department, Boston University
created[InstanceEdit:352922] D'Eustachio, P, 2008-06-09 12:49:16
crossReference[DatabaseIdentifier:5694819] ORCID:0000-0002-0598-7241
firstnameDean R
initialDR
modified[InstanceEdit:469706] D'Eustachio, P, 2010-01-25
[InstanceEdit:5656436] D'Eustachio, Peter, 2014-12-12
[InstanceEdit:5675813] D'Eustachio, Peter, 2015-02-16
[InstanceEdit:5694820] D'Eustachio, Peter, 2015-05-23
surnameTolan
(author)[InstanceEdit:5675821] Timson, David J, Tolan, Dean R, 2015-02-16
[LiteratureReference:352901] Noncoordinate changes in the steady-state mRNA expressed from aldolase A and aldolase C genes during differentiation of chicken myoblasts
[LiteratureReference:469704] Molecular basis of hereditary fructose intolerance: mutations and polymorphisms in the human aldolase B gene
[LiteratureReference:469723] The structure of human liver fructose-1,6-bisphosphate aldolase
[LiteratureReference:5656437] Catalytic deficiency of human aldolase B in hereditary fructose intolerance caused by a common missense mutation
[LiteratureReference:5675278] Molecular analysis of aldolase B genes in hereditary fructose intolerance
[LiteratureReference:5675282] The temperature dependence of activity and structure for the most prevalent mutant aldolase B associated with hereditary fructose intolerance
[LiteratureReference:5675309] Genes required for fructose metabolism are expressed in Purkinje cells in the cerebellum
[LiteratureReference:5675313] Increased prevalence of mutant null alleles that cause hereditary fructose intolerance in the American population
[LiteratureReference:5675318] Aldolase-B knockout in mice phenocopies hereditary fructose intolerance in humans
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