Reactome: A Curated Pathway Database

Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

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Pathways reviewed by Grummt, Ingrid (5084141)

DB_ID Name
427359 SIRT1 negatively regulates rRNA expression

Details on Person Grummt, Ingrid

_displayNameGrummt, Ingrid
_timestamp2017-08-22 20:44:58
created[InstanceEdit:5084143] May, B, 2013-11-08
(author)[InstanceEdit:5246500] Voit, Renate, Grummt, Ingrid, 2014-01-21
[LiteratureReference:5084145] Intergenic transcripts originating from a subclass of ribosomal DNA repeats silence ribosomal RNA genes in trans
[LiteratureReference:5096457] Epigenetic control of RNA polymerase I transcription in mammalian cells
[LiteratureReference:5096460] The chromatin remodeling complex NuRD establishes the poised state of rRNA genes characterized by bivalent histone modifications and altered nucleosome positions
[LiteratureReference:5096481] Interaction of noncoding RNA with the rDNA promoter mediates recruitment of DNMT3b and silencing of rRNA genes
[LiteratureReference:5211368] Chromatin-specific regulation of mammalian rDNA transcription by clustered TTF-I binding sites
[LiteratureReference:5219065] Recruitment of the nucleolar remodeling complex NoRC establishes ribosomal DNA silencing in chromatin
[LiteratureReference:5219066] The chromatin remodeling complex NoRC and TTF-I cooperate in the regulation of the mammalian rRNA genes in vivo
[LiteratureReference:5246498] Linking rDNA transcription to the cellular energy supply
[LiteratureReference:5250940] The chromatin remodelling complex WSTF-SNF2h interacts with nuclear myosin 1 and has a role in RNA polymerase I transcription
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