Resting potential and submembrane calcium concentration of inner hair cells in the isolated mouse cochlea are set by KCNQ-type potassium channels
KCNQ4, a K+ channel mutated in a form of dominant deafness, is expressed in the inner ear and the central auditory pathway
|Proc. Natl. Acad. Sci. U.S.A.
Thyroid hormone receptors TRalpha1 and TRbeta differentially regulate gene expression of Kcnq4 and prestin during final differentiation of outer hair cells
|J. Cell. Sci.
Longitudinal gradients of KCNQ4 expression in spiral ganglion and cochlear hair cells correlate with progressive hearing loss in DFNA2
|Brain Res. Mol. Brain Res.
Mice with altered KCNQ4 K+ channels implicate sensory outer hair cells in human progressive deafness
Differential expression of KCNQ4 in inner hair cells and sensory neurons is the basis of progressive high-frequency hearing loss
Roles of alternative splicing in the functional properties of inner ear-specific KCNQ4 channels
|J. Biol. Chem.